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Recent related articles ( identify beta):

Researchers identify molecule that tells your brain your stomach is full
In studies with mice and rats, researchers have found that a chemical messenger called NAPE is made in the small intestine after the animals ate a greasy meal. After eating, NAPE - N-acylphosphatidylethanolamine, a mouthful in itself -- enters the blood and travels to the brain, where it
Researchers identify molecule that tells your brain your stomach is full...

Presence of gum disease may help dentists and physicians identify risk for cardiovascular disease
Individuals reporting a history of periodontal disease were more likely to have increased levels of inflammation, a risk factor for heart disease, compared to those who reported no history of periodontal disease, according to an American Journal of Cardiology report available online today.
Presence of gum disease may help dentists and physicians identify risk for cardiovascular disease...

Interferon-[beta]-1b better for chronic viral cardiomyopathy.
Page: 6 (Source: Inpharma Weekly)
Interferon-[beta]-1b better for chronic viral cardiomyopathy....

Cutting edge: selective blockade of light-lymphotoxin {beta} receptor signaling protects mice from experimental cerebral malaria caused by plasmodium berghei anka.
In this study we report a role for LIGHT-lymphotoxin beta Receptor (LTbetaR) signaling in the development of ECM and control of parasite growth. Specific blockade of LIGHT-LTbetaR, but not LIGHT-herpesvirus entry mediator interactions, abrogated the accumulation of parasites and the recruitment of pathogenic CD8(+) T cells and monocytes
Cutting edge: selective blockade of light-lymphotoxin {beta} receptor signaling protects mice from experimental cerebral malaria caused by plasmodium berghei anka....

Ifn-beta production by tlr4-stimulated innate immune cells is negatively regulated by gsk3-beta.
In this study, we demonstrate glycogen synthase kinase 3-beta (GSK3-beta) plays a fundamental role in this process. Suppression of GSK3-beta activity by either pharmacological inhibition, small interfering RNA-mediated gene silencing, or ectopic expression of a kinase-dead GSK3-beta mutant enhanced IFN-beta production by TLR4-stimulated macrophages. Conversely, ectopic expression of
Ifn-beta production by tlr4-stimulated innate immune cells is negatively regulated by gsk3-beta....

Deletion of gsk-3{beta} in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation
We report that terminal cardiomyocyte differentiation was substantially blunted in Gsk3b–/– embryoid bodies. While GSK-3α–deficient mice were born without a cardiac phenotype, no live-born Gsk3b–/– pups were recovered. The Gsk3b–/– embryos had a double outlet RV, ventricular septal defects, and hypertrophic myopathy, with near obliteration of the ventricular
Deletion of gsk-3{beta} in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation...

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