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Recent related articles ( interfere beta 1a):

Interferon-[beta]-1b better for chronic viral cardiomyopathy.
Page: 6 (Source: Inpharma Weekly)
Interferon-[beta]-1b better for chronic viral cardiomyopathy....

Cutting edge: selective blockade of light-lymphotoxin {beta} receptor signaling protects mice from experimental cerebral malaria caused by plasmodium berghei anka.
In this study we report a role for LIGHT-lymphotoxin beta Receptor (LTbetaR) signaling in the development of ECM and control of parasite growth. Specific blockade of LIGHT-LTbetaR, but not LIGHT-herpesvirus entry mediator interactions, abrogated the accumulation of parasites and the recruitment of pathogenic CD8(+) T cells and monocytes
Cutting edge: selective blockade of light-lymphotoxin {beta} receptor signaling protects mice from experimental cerebral malaria caused by plasmodium berghei anka....

Ifn-beta production by tlr4-stimulated innate immune cells is negatively regulated by gsk3-beta.
In this study, we demonstrate glycogen synthase kinase 3-beta (GSK3-beta) plays a fundamental role in this process. Suppression of GSK3-beta activity by either pharmacological inhibition, small interfering RNA-mediated gene silencing, or ectopic expression of a kinase-dead GSK3-beta mutant enhanced IFN-beta production by TLR4-stimulated macrophages. Conversely, ectopic expression of
Ifn-beta production by tlr4-stimulated innate immune cells is negatively regulated by gsk3-beta....

Deletion of gsk-3{beta} in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation
We report that terminal cardiomyocyte differentiation was substantially blunted in Gsk3b–/– embryoid bodies. While GSK-3α–deficient mice were born without a cardiac phenotype, no live-born Gsk3b–/– pups were recovered. The Gsk3b–/– embryos had a double outlet RV, ventricular septal defects, and hypertrophic myopathy, with near obliteration of the ventricular
Deletion of gsk-3{beta} in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation...

Reversal of iron-induced dilated cardiomyopathy during therapy with deferasirox in beta-thalassemia
A 15-year-old male with [beta]-thalassemia major developed dilated cardiomyopathy secondary to iron-overload (Z-scores of left ventricle (LV) dimensions >3, ejection fraction (EF) 33%). Treatment with deferoxamine was unsuccessful, presumably due to poor compliance. After 15 months of using deferasirox (DFX), LV end-diastolic dimension normalized (Z-scores (Source: Pediatric Blood
Reversal of iron-induced dilated cardiomyopathy during therapy with deferasirox in beta-thalassemia...

A phosphatidylserine species inhibits a range of tlr- but not il-1beta-induced inflammatory responses by disruption of membrane microdomains.
Related Articles A phosphatidylserine species inhibits a range of TLR- but not IL-1beta-induced inflammatory responses by disruption of membrane microdomains. J Immunol. 2008 Oct 15;181(8):5606-17
A phosphatidylserine species inhibits a range of tlr- but not il-1beta-induced inflammatory responses by disruption of membrane microdomains....

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